David Lafont
Impact in
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- Congenital limb and hand anomalies
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- Neurogenesis and neuroplasticity mechanisms
Papers in
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- Protein Tyrosine Phosphatases 3
- Hedgehog Signaling Pathway Studies 1
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- Neuroinflammation and Neurodegeneration Mechanisms 2
- Co-authors
- Paola Zaratin (3 shared papers)Béatrice Gréco (3 shared papers)Tiziana Adage (1 shared paper)Edward J. Ryder (1 shared paper)Sarah Spiegel (1 shared paper)Christopher J. Lelliott (1 shared paper)Jeffrey K. Huang (1 shared paper)Robin J.M. Franklin (1 shared paper)
- Journals
- American Journal Of Pathology (1 paper)Journal of Bone and Mineral Research (1 paper)Journal of Inflammation (1 paper)Behavioural Brain Research (1 paper)The Journal of Pathology (1 paper)
- Partner nations
- United KingdomUnited StatesSwitzerland
In The Last Decade
David Lafont
5 papers receiving 105 citations
Peers
Comparison fields: 5 of 42
- Developmental Biology 13
- Developmental Neuroscience 10
- Neurology 11
- Cell Biology 19
- Immunology 21
Countries citing papers authored by David Lafont
This map shows the geographic impact of David Lafont's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Lafont with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Lafont more than expected).
Fields of papers citing papers by David Lafont
This network shows the impact of papers produced by David Lafont. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Lafont. The network helps show where David Lafont may publish in the future.
Co-authors
The 25 scholars most cited alongside David Lafont, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2016 | 34 | |
| 2 | 2018 | 25 | |
| 3 | 2009 | 24 | |
| 4 | 2012 | 17 | |
| 5 | 2010 | 5 | |
| 6 | 2025 | 0 |
About David Lafont
David Lafont is a scholar working on Molecular Biology, Neurology, Surgery, Developmental Biology and Cell Biology, having authored 6 papers that have together received 105 indexed citations. Recurring topics across this work include Protein Tyrosine Phosphatases (3 papers), Neuroinflammation and Neurodegeneration Mechanisms (2 papers), Neurogenesis and neuroplasticity mechanisms (1 paper), Hedgehog Signaling Pathway Studies (1 paper), Endoplasmic Reticulum Stress and Disease (1 paper), Congenital limb and hand anomalies (1 paper), Cardiovascular, Neuropeptides, and Oxidative Stress Research (1 paper) and Connective tissue disorders research (1 paper). The work is most often cited by research in Developmental Biology (13 citations), Developmental Neuroscience (10 citations), Neurology (11 citations), Cell Biology (19 citations) and Immunology (21 citations). David Lafont has collaborated with scholars based in United Kingdom, United States and Switzerland. Frequent co-authors include Paola Zaratin, Béatrice Gréco, Tiziana Adage, Edward J. Ryder, Sarah Spiegel, Christopher J. Lelliott, Jeffrey K. Huang, Robin J.M. Franklin, Carina Cintia Ferrari and Mark J. Arends. Their work appears in journals such as American Journal Of Pathology, Journal of Bone and Mineral Research, Journal of Inflammation, Behavioural Brain Research and The Journal of Pathology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.