David Jay

599 citations
25 papers · 528 · h-index 14

Impact in

Papers in

    • Mitochondrial Function and Pathology 4
    • Signaling Pathways in Disease 4
    • Calpain Protease Function and Regulation 3
    • Cellular Mechanics and Interactions 2

David Jay

24 papers receiving 516 citations

Peers

David Jay
Comparison fields: 5 of 89
  • Cell Biology 115
  • Health, Toxicology and Mutagenesis 81
  • Nutrition and Dietetics 76
  • Immunology and Allergy 20
  • Molecular Biology 214
Replace Michael A. Hass with:
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Citations per field
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Citations per year

Countries citing papers authored by David Jay

Since Specialization
Citations

This map shows the geographic impact of David Jay's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Jay with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Jay more than expected).

Fields of papers citing papers by David Jay

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David Jay. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Jay. The network helps show where David Jay may publish in the future.

Co-authors

The 24 scholars most cited alongside David Jay, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with David Jay Line = papers co-authored together David Jay links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 25 papers — load more, or switch the sort, to bring in the rest.

#Work
1 1985101
2 200756
3 200049
4 200438
5 198736
6 200534
7 199130
8 199130
9 199621
10 199917
11 199517
12 198716
13 199716
14 201013
15 200710
16 199110
17 19928
18 19948
19
Captopril and glutathione inhibit the superoxide dismutase activity of Hg (II).
19995
20 20024

About David Jay

David Jay is a scholar working on Molecular Biology, Cell Biology, Biochemistry, Health, Toxicology and Mutagenesis and Physiology, having authored 25 papers that have together received 528 indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (4 papers), Signaling Pathways in Disease (4 papers), Calpain Protease Function and Regulation (3 papers), Electrochemical Analysis and Applications (3 papers), Biochemical Acid Research Studies (2 papers), Trace Elements in Health (2 papers), Sulfur Compounds in Biology (2 papers) and Cellular Mechanics and Interactions (2 papers). The work is most often cited by research in Cell Biology (115 citations), Health, Toxicology and Mutagenesis (81 citations), Nutrition and Dietetics (76 citations), Immunology and Allergy (20 citations) and Molecular Biology (214 citations). David Jay has collaborated with scholars based in Mexico, Brazil and United States. Frequent co-authors include Edmundo Chávez, Luz Ibarra-Lara, Concepción Bravo, Alfred Stracher, Miguel Ángel Medina, Elizabeth García, R. Zamorano, Eduardo Muñóz, J. L. Boldú and Leticia Bucio. Their work appears in journals such as Journal of Bioenergetics and Biomembranes, Archives of Biochemistry and Biophysics, Biochemical and Biophysical Research Communications, Molecular and Cellular Biochemistry and Archives of Medical Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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