David E. Magee

17 papers receiving 1.4k citations

David E. Magee's Hit Papers

Raloxifene (LY139481 HCI) prevents bone loss and reduces serum cholesterol without causing uterine hypertrophy in ovariectomized rats. 1994 · 552 citations
5520+10+21Years since publication100200300400500

Peers

David E. Magee
Comparison fields: 5 of 82
  • Genetics 880
  • Orthopedics and Sports Medicine 215
  • Toxicology 78
  • Endocrinology, Diabetes and Metabolism 352
  • Reproductive Medicine 99
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Citations per field
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Citations per year

Countries citing papers authored by David E. Magee

Since Specialization
Citations

This map shows the geographic impact of David E. Magee's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David E. Magee with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David E. Magee more than expected).

Fields of papers citing papers by David E. Magee

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David E. Magee. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David E. Magee. The network helps show where David E. Magee may publish in the future.

Co-authors

The 25 scholars most cited alongside David E. Magee, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with David E. Magee Line = papers co-authored together David E. Magee links everyone, so they are left out of the graph.

All Works

17 of 17 papers shown
#Work
1
Raloxifene (LY139481 HCI) prevents bone loss and reduces serum cholesterol without causing uterine hypertrophy in ovariectomized rats.
Hit paper breakdown →
1994552
2 1997158
3 1996136
4 1998121
5 199498
6 199489
7 199578
8 199670
9 199334
10 199732
11 199718
12 199617
13 199515
14 199112
15 199611
16 200310
17 19967

About David E. Magee

David E. Magee is a scholar working on Genetics, Endocrinology, Diabetes and Metabolism, Molecular Biology, Orthopedics and Sports Medicine and Organic Chemistry, having authored 17 papers that have together received 1.5k indexed citations. Recurring topics across this work include Estrogen and related hormone effects (13 papers), Bone health and osteoporosis research (4 papers), Menopause: Health Impacts and Treatments (4 papers), Endometriosis Research and Treatment (3 papers), Endometrial and Cervical Cancer Treatments (2 papers), Bioactive Compounds and Antitumor Agents (2 papers), Inflammatory mediators and NSAID effects (2 papers) and Genomics, phytochemicals, and oxidative stress (2 papers). The work is most often cited by research in Genetics (880 citations), Orthopedics and Sports Medicine (215 citations), Toxicology (78 citations), Endocrinology, Diabetes and Metabolism (352 citations) and Reproductive Medicine (99 citations). David E. Magee has collaborated with scholars based in United States. Frequent co-authors include Masahiko Sato, Ellen R. Rowley, Henry U. Bryant, George J. Cullinan, Raymond F. Kauffman, A Bekele, Larry J. Black, D. Clive Williams, William R. Bensch and Raymond A. Bendele. Their work appears in journals such as Bioorganic & Medicinal Chemistry Letters, Endocrinology, Journal of Medicinal Chemistry, Journal of Clinical Investigation and Molecular Pharmacology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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