David Duguay

488 citations
11 papers · 415 · h-index 9

Impact in

Papers in

David Duguay

11 papers receiving 410 citations

Peers

David Duguay
Comparison fields: 5 of 78
  • Endocrine and Autonomic Systems 232
  • Aging 43
  • Physiology 115
  • Biological Psychiatry 9
  • Experimental and Cognitive Psychology 37
Replace Edward A. Hayter with:
Edward A. Hayter United Kingdom
Sarah McLoughlin Ireland
Maoxiang Zhang United States
Irène Yujnovsky France
Pascale Bouchard-Cannon Canada
Benjamin Ladeuix Israel
Jeremy J. Stubblefield United States
Andrea Brenna Switzerland
Naohiro Kon Japan
Melissa A. Wren United States
David Duguay relative to Edward A. Hayter United Kingdom Edward A. Hayter's profile →
Citations per field
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Edward A. Hayter · 1×
Citations per year

Countries citing papers authored by David Duguay

Since Specialization
Citations

This map shows the geographic impact of David Duguay's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Duguay with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Duguay more than expected).

Fields of papers citing papers by David Duguay

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David Duguay. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Duguay. The network helps show where David Duguay may publish in the future.

Co-authors

The 25 scholars most cited alongside David Duguay, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with David Duguay Line = papers co-authored together David Duguay links everyone, so they are left out of the graph.

All Works

11 of 11 papers shown
#Work
1 2009178
2 201185
3 201237
4 201434
5 201123
6 200717
7 200413
8 200711
9 200711
10 20044
11 20152

About David Duguay

David Duguay is a scholar working on Endocrine and Autonomic Systems, Molecular Biology, Cardiology and Cardiovascular Medicine, Physiology and Plant Science, having authored 11 papers that have together received 415 indexed citations. Recurring topics across this work include Circadian rhythm and melatonin (5 papers), Coenzyme Q10 studies and effects (2 papers), Renin-Angiotensin System Studies (2 papers), Adolescent and Pediatric Healthcare (1 paper), Nitric Oxide and Endothelin Effects (1 paper), Epigenetics and DNA Methylation (1 paper), Coagulation, Bradykinin, Polyphosphates, and Angioedema (1 paper) and Diet, Metabolism, and Disease (1 paper). The work is most often cited by research in Endocrine and Autonomic Systems (232 citations), Aging (43 citations), Physiology (115 citations), Biological Psychiatry (9 citations) and Experimental and Cognitive Psychology (37 citations). David Duguay has collaborated with scholars based in Canada, Denmark and United States. Frequent co-authors include Nicolas Cermakian, Denis deBlois, Simon S. Wing, Jan Fahrenkrug, Catherine R. Dufour, Brian J. Wilson, Vincent Giguère, Lillian J. Eichner, Armen Khatchadourian and Valérie Mongrain. Their work appears in journals such as British Journal of Pharmacology, Biology Open, Journal of Biological Rhythms, Journal of Hypertension and PLoS ONE.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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