Daniel Wynn

48 papers receiving 3.5k citations

Daniel Wynn's Hit Papers

Natalizumab plus Interferon Beta-1a for Relapsing Multiple Sclerosis 2006 · 982 citations
9820+6+13Years since publication250500750

Peers

Daniel Wynn
Comparison fields: 5 of 116
  • Pathology and Forensic Medicine 2.7k
  • Neurology 1.1k
  • Rheumatology 653
  • Immunology 792
  • Oncology 1.0k
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Daniel Wynn relative to Jeffrey Cohen United States Jeffrey Cohen's profile →
Citations per field
00.5×1.5×
Jeffrey Cohen · 1×
Citations per year

Countries citing papers authored by Daniel Wynn

Since Specialization
Citations

This map shows the geographic impact of Daniel Wynn's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daniel Wynn with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daniel Wynn more than expected).

Fields of papers citing papers by Daniel Wynn

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Daniel Wynn. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daniel Wynn. The network helps show where Daniel Wynn may publish in the future.

Co-authors

The 25 scholars most cited alongside Daniel Wynn, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Daniel Wynn Line = papers co-authored together Daniel Wynn links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 50 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Natalizumab plus Interferon Beta-1a for Relapsing Multiple Sclerosis
Hit paper breakdown →
2006982
2 2009407
3 2007236
4 2010234
5 2009183
6 1990149
7 2010147
8 2006126
9 2022111
10 2011101
11 201093
12 201280
13 200478
14 201466
15 201059
16 201550
17 201050
18 201043
19 201042
20 201140

About Daniel Wynn

Daniel Wynn is a scholar working on Pathology and Forensic Medicine, Neurology, Immunology, Oncology and Hematology, having authored 50 papers that have together received 3.6k indexed citations. Recurring topics across this work include Multiple Sclerosis Research Studies (44 papers), Peripheral Neuropathies and Disorders (10 papers), Autoimmune and Inflammatory Disorders Research (8 papers), Polyomavirus and related diseases (7 papers), Immunotherapy and Immune Responses (7 papers), Rheumatoid Arthritis Research and Therapies (5 papers), Monoclonal and Polyclonal Antibodies Research (5 papers) and Powdery Mildew Fungal Diseases (4 papers). The work is most often cited by research in Pathology and Forensic Medicine (2.7k citations), Neurology (1.1k citations), Rheumatology (653 citations), Immunology (792 citations) and Oncology (1.0k citations). Daniel Wynn has collaborated with scholars based in United States, Germany and Poland. Frequent co-authors include Steven Galetta, Fred Lublin, Christian Confavreux, Frances Lynn, Peter A. Calabresi, Michael Panzara, Richard A. Rudick, Alfred Sandrock, Bianca Weinstock‐Guttman and Ernst-Wilhelm Radüe. Their work appears in journals such as Neurology, Multiple Sclerosis and Related Disorders, Multiple Sclerosis Journal, Annals of Neurology and Journal of the Neurological Sciences.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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