Daniel Perdiz
Impact in
- Physiology top 2%
- Dermatology top 1%
- Skin Protection and Aging
Papers in
-
- DNA Repair Mechanisms 5
- DNA and Nucleic Acid Chemistry 3
- Ubiquitin and proteasome pathways 2
-
- Autophagy in Disease and Therapy 6
- Co-authors
- Christian Poüs (9 shared papers)Évelyne Sage (5 shared papers)Rafah Mackeh (3 shared papers)Sara L. Tamisier‐Karolak (2 shared papers)Y. Lévi (1 shared paper)Anita Baillet (3 shared papers)Patrice Codogno (3 shared papers)Pál Gróf (3 shared papers)
In The Last Decade
Daniel Perdiz
20 papers receiving 1.9k citations
Peers
Comparison fields: 5 of 113
- Physiology 146
- Dermatology 258
- Pollution 332
- Health, Toxicology and Mutagenesis 329
- Cell Biology 291
Countries citing papers authored by Daniel Perdiz
This map shows the geographic impact of Daniel Perdiz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daniel Perdiz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daniel Perdiz more than expected).
Fields of papers citing papers by Daniel Perdiz
This network shows the impact of papers produced by Daniel Perdiz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daniel Perdiz. The network helps show where Daniel Perdiz may publish in the future.
Co-authors
The 25 scholars most cited alongside Daniel Perdiz, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2004 | 336 | |
| 2 | 2000 | 209 | |
| 3 | 2000 | 197 | |
| 4 | 1999 | 193 | |
| 5 | 2010 | 177 | |
| 6 | 2013 | 165 | |
| 7 | 2010 | 155 | |
| 8 | 1999 | 100 | |
| 9 | 2014 | 77 | |
| 10 | 2020 | 76 | |
| 11 | 2008 | 61 | |
| 12 | 2003 | 44 | |
| 13 | 2017 | 34 | |
| 14 | 2006 | 20 | |
| 15 | 2019 | 18 | |
| 16 | 2022 | 17 | |
| 17 | 2005 | 12 | |
| 18 | 1999 | 12 | |
| 19 | 2006 | 9 | |
| 20 | 1997 | 2 |
About Daniel Perdiz
Daniel Perdiz is a scholar working on Molecular Biology, Epidemiology, Health, Toxicology and Mutagenesis, Cell Biology and Pollution, having authored 20 papers that have together received 1.9k indexed citations. Recurring topics across this work include Autophagy in Disease and Therapy (6 papers), DNA Repair Mechanisms (5 papers), Effects and risks of endocrine disrupting chemicals (4 papers), Carcinogens and Genotoxicity Assessment (3 papers), DNA and Nucleic Acid Chemistry (3 papers), Pharmaceutical and Antibiotic Environmental Impacts (3 papers), Ubiquitin and proteasome pathways (2 papers) and Endoplasmic Reticulum Stress and Disease (2 papers). The work is most often cited by research in Physiology (146 citations), Dermatology (258 citations), Pollution (332 citations), Health, Toxicology and Mutagenesis (329 citations) and Cell Biology (291 citations). Daniel Perdiz has collaborated with scholars based in France, Hungary and Italy. Frequent co-authors include Christian Poüs, Évelyne Sage, Rafah Mackeh, Sara L. Tamisier‐Karolak, Y. Lévi, Anita Baillet, Patrice Codogno, Pál Gróf, E. Moustacchi and Séverine Lorin. Their work appears in journals such as Journal of Biological Chemistry, Cellular Signalling, Journal of Photochemistry and Photobiology B Biology, Photochemistry and Photobiology and Journal of Cell Science.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.