Daniel Militz

490 citations
6 papers · 383 · h-index 5

Impact in

  • Cell Biology top 10%
    • Cellular transport and secretion
    • Endoplasmic Reticulum Stress and Disease
    • Alzheimer's disease research and treatments

Papers in

Daniel Militz

6 papers receiving 378 citations

Peers

Daniel Militz
Comparison fields: 5 of 59
  • Cell Biology 160
  • Physiology 185
  • Developmental Neuroscience 21
  • Cellular and Molecular Neuroscience 78
  • Physiology 15
Replace Lilia Rodriguez with:
Lilia Rodriguez Argentina
Francisco Llavero Spain
Ditte Olsen Denmark
Mailis Liiv Estonia
Zhejun Ji China
Dae-Hoon Lee South Korea
Akbar Zeb Estonia
Yon Ju Ji United States
Hideaki Asahara Japan
Chul-Woong Chung South Korea
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Citations per field
00.5×2.6×
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Citations per year

Countries citing papers authored by Daniel Militz

Since Specialization
Citations

This map shows the geographic impact of Daniel Militz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daniel Militz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daniel Militz more than expected).

Fields of papers citing papers by Daniel Militz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Daniel Militz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daniel Militz. The network helps show where Daniel Militz may publish in the future.

Co-authors

The 25 scholars most cited alongside Daniel Militz, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Daniel Militz Line = papers co-authored together Daniel Militz links everyone, so they are left out of the graph.

All Works

6 of 6 papers shown
#Work
1 2012203
2 200884
3 200246
4 201044
5 20244
6 20212

About Daniel Militz

Daniel Militz is a scholar working on Cellular and Molecular Neuroscience, Endocrinology, Diabetes and Metabolism, Cell Biology, Physiology and Surgery, having authored 6 papers that have together received 383 indexed citations. Recurring topics across this work include Diabetes Management and Research (2 papers), Alzheimer's disease research and treatments (2 papers), Pancreatic function and diabetes (1 paper), Ion channel regulation and function (1 paper), Diabetes, Cardiovascular Risks, and Lipoproteins (1 paper), Neurobiology and Insect Physiology Research (1 paper), Diabetes Management and Education (1 paper) and Vitamin D Research Studies (1 paper). The work is most often cited by research in Cell Biology (160 citations), Physiology (185 citations), Developmental Neuroscience (21 citations), Cellular and Molecular Neuroscience (78 citations) and Physiology (15 citations). Daniel Militz has collaborated with scholars based in United States, Germany and United Kingdom. Frequent co-authors include Thomas E. Willnow, Vanessa Schmidt, Olav M. Andersen, Anders Nykjær, Erik Christensen, Chengbiao Wu, Camilla Gustafsen, Suzanne Gokool, Matthew Seaman and Peder Madsen. Their work appears in journals such as Journal of Cell Science, Journal of Diabetes Science and Technology, Journal of Neuroscience, Journal of Biological Chemistry and Molecular and Cellular Biology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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