D. Kirsch
Impact in
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- Metabolism, Diabetes, and Cancer
- Protein Kinase Regulation and GTPase Signaling
- Receptor Mechanisms and Signaling
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- Growth Hormone and Insulin-like Growth Factors
- Diabetes Treatment and Management
Papers in
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- Metabolism, Diabetes, and Cancer 11
- Peroxisome Proliferator-Activated Receptors 1
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- Adipose Tissue and Metabolism 1
- Co-authors
- B. Obermaier (6 shared papers)Hans‐Ulrich Häring (5 shared papers)Fausto E Machicao (6 shared papers)Britta Ermel (5 shared papers)Hans-Ulrich Häring (3 shared papers)WOLFGANG K. KEMMLER (3 shared papers)Franz Rinninger (3 shared papers)Thomas Deufel (1 shared paper)
- Journals
- FEBS Letters (3 papers)Biochemical and Biophysical Research Communications (2 papers)Biochemical Journal (2 papers)Diabetologia (2 papers)Journal of Biological Chemistry (1 paper)
- Partner nations
- GermanyUnited States
In The Last Decade
D. Kirsch
11 papers receiving 446 citations
Peers
Comparison fields: 5 of 37
- Molecular Biology 352
- Endocrinology, Diabetes and Metabolism 93
- Biochemistry 22
- Physiology 44
- Physiology 4
Countries citing papers authored by D. Kirsch
This map shows the geographic impact of D. Kirsch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by D. Kirsch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites D. Kirsch more than expected).
Fields of papers citing papers by D. Kirsch
This network shows the impact of papers produced by D. Kirsch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by D. Kirsch. The network helps show where D. Kirsch may publish in the future.
Co-authors
The 16 scholars most cited alongside D. Kirsch, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 1986 | 105 | |
| 2 | 1986 | 88 | |
| 3 | 1985 | 71 | |
| 4 | 1983 | 60 | |
| 5 | 1987 | 36 | |
| 6 | 1983 | 34 | |
| 7 | 1984 | 29 | |
| 8 | 1984 | 21 | |
| 9 | Insulin receptor kinase defects as a possible cause of cellular insulin resistance. | 1987 | 17 |
| 10 | 1985 | 12 | |
| 11 | 1984 | 10 |
About D. Kirsch
D. Kirsch is a scholar working on Molecular Biology, Physiology, Surgery, Nuclear Energy and Engineering and Waste Management and Disposal, having authored 11 papers that have together received 483 indexed citations. Recurring topics across this work include Metabolism, Diabetes, and Cancer (11 papers), Pancreatic function and diabetes (1 paper), Peroxisome Proliferator-Activated Receptors (1 paper) and Adipose Tissue and Metabolism (1 paper). The work is most often cited by research in Molecular Biology (352 citations), Endocrinology, Diabetes and Metabolism (93 citations), Biochemistry (22 citations), Physiology (44 citations) and Physiology (4 citations). D. Kirsch has collaborated with scholars based in Germany and United States. Frequent co-authors include B. Obermaier, Hans‐Ulrich Häring, Fausto E Machicao, Britta Ermel, Hans-Ulrich Häring, WOLFGANG K. KEMMLER, Franz Rinninger, Thomas Deufel, W. E. Bachmann and Hans Ulrich Häring. Their work appears in journals such as FEBS Letters, Biochemical and Biophysical Research Communications, Biochemical Journal, Diabetologia and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.