Cleta D’Sa-Eipper
Impact in
- Developmental Neuroscience top 5%
- Neurogenesis and neuroplasticity mechanisms
- Molecular Biology top 10%
- Cell death mechanisms and regulation
- Mitochondrial Function and Pathology
- RNA Interference and Gene Delivery
- Ubiquitin and proteasome pathways
Papers in
-
- Cell death mechanisms and regulation 8
- Viral Infectious Diseases and Gene Expression in Insects 1
- DNA Repair Mechanisms 1
- Ubiquitin and proteasome pathways 1
- Fungal and yeast genetics research 1
- Oncology 3
- Cancer-related Molecular Pathways 3
- Co-authors
- G. Chinnadurai (7 shared papers)T. Subramanian (4 shared papers)Kevin A. Roth (4 shared papers)Ute Schaeper (1 shared paper)Scott Malstrom (1 shared paper)Leelavathi Venkatesh (1 shared paper)Tejas Subramanian (1 shared paper)Keisuke Kuida (3 shared papers)
- Journals
- Oncogene (2 papers)Developmental Neuroscience (1 paper)Development (1 paper)Cell (1 paper)Proceedings of the National Academy of Sciences (1 paper)
- Partner nations
- United States
In The Last Decade
Cleta D’Sa-Eipper
11 papers receiving 1.0k citations
Peers
Comparison fields: 5 of 81
- Developmental Neuroscience 84
- Molecular Biology 765
- Cancer Research 140
- Oncology 216
- Immunology 162
Countries citing papers authored by Cleta D’Sa-Eipper
This map shows the geographic impact of Cleta D’Sa-Eipper's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Cleta D’Sa-Eipper with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Cleta D’Sa-Eipper more than expected).
Fields of papers citing papers by Cleta D’Sa-Eipper
This network shows the impact of papers produced by Cleta D’Sa-Eipper. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Cleta D’Sa-Eipper. The network helps show where Cleta D’Sa-Eipper may publish in the future.
Co-authors
The 21 scholars most cited alongside Cleta D’Sa-Eipper, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 1994 | 422 | |
| 2 | 2001 | 111 | |
| 3 | 2000 | 102 | |
| 4 | bfl-1, a bcl-2 homologue, suppresses p53-induced apoptosis and exhibits potent cooperative transforming activity. | 1996 | 97 |
| 5 | 2001 | 65 | |
| 6 | Deletion of a nonconserved region of Bcl-2 confers a novel gain of function: suppression of apoptosis with concomitant cell proliferation. | 1996 | 63 |
| 7 | 1998 | 61 | |
| 8 | Unmasking of a proliferation-restraining activity of the anti-apoptosis protein EBV BHRF1. | 1996 | 50 |
| 9 | 2000 | 46 | |
| 10 | 1998 | 29 | |
| 11 | 1994 | 8 |
About Cleta D’Sa-Eipper
Cleta D’Sa-Eipper is a scholar working on Molecular Biology, Oncology, Cancer Research, Infectious Diseases and Virology, having authored 11 papers that have together received 1.1k indexed citations. Recurring topics across this work include Cell death mechanisms and regulation (8 papers), Cancer-related Molecular Pathways (3 papers), MicroRNA in disease regulation (3 papers), Viral Infectious Diseases and Gene Expression in Insects (1 paper), Neurogenesis and neuroplasticity mechanisms (1 paper), DNA Repair Mechanisms (1 paper), Ubiquitin and proteasome pathways (1 paper) and Fungal and yeast genetics research (1 paper). The work is most often cited by research in Developmental Neuroscience (84 citations), Molecular Biology (765 citations), Cancer Research (140 citations), Oncology (216 citations) and Immunology (162 citations). Cleta D’Sa-Eipper has collaborated with scholars based in United States. Frequent co-authors include G. Chinnadurai, T. Subramanian, Kevin A. Roth, Ute Schaeper, Scott Malstrom, Leelavathi Venkatesh, Tejas Subramanian, Keisuke Kuida, Pasko Rakić and Richard A. Flavell. Their work appears in journals such as Oncogene, Developmental Neuroscience, Development, Cell and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.