Chisa Okada
Impact in
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- Gout, Hyperuricemia, Uric Acid
Papers in
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- RNA Research and Splicing 3
- CRISPR and Genetic Engineering 3
- RNA regulation and disease 2
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- Autophagy in Disease and Therapy 6
- Co-authors
- Hideo Tsukamoto (2 shared papers)Noboru Kawabe (1 shared paper)Motoki Osawa (1 shared paper)Yu Kakimoto (1 shared paper)Hitoshi Endo (3 shared papers)Masayuki Tatemichi (3 shared papers)Satoshi Owada (3 shared papers)Akira T. Kawaguchi (1 shared paper)
- Journals
- PLoS ONE (2 papers)Anticancer Research (2 papers)Scientific Reports (2 papers)Biochemical and Biophysical Research Communications (1 paper)iScience (1 paper)
- Partner nations
- JapanSwitzerlandThailand
In The Last Decade
Chisa Okada
20 papers receiving 273 citations
Peers
Comparison fields: 5 of 72
- Aging 4
- Nephrology 13
- Physiology 9
- Cancer Research 27
- Molecular Biology 121
Countries citing papers authored by Chisa Okada
This map shows the geographic impact of Chisa Okada's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Chisa Okada with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Chisa Okada more than expected).
Fields of papers citing papers by Chisa Okada
This network shows the impact of papers produced by Chisa Okada. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Chisa Okada. The network helps show where Chisa Okada may publish in the future.
Co-authors
The 25 scholars most cited alongside Chisa Okada, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2019 | 66 | |
| 2 | 2018 | 26 | |
| 3 | 2011 | 22 | |
| 4 | 2021 | 17 | |
| 5 | 2015 | 15 | |
| 6 | 2020 | 14 | |
| 7 | 2019 | 13 | |
| 8 | 2018 | 12 | |
| 9 | 2021 | 11 | |
| 10 | 2017 | 11 | |
| 11 | 1999 | 10 | |
| 12 | 2022 | 10 | |
| 13 | 2011 | 9 | |
| 14 | 2020 | 9 | |
| 15 | 2017 | 8 | |
| 16 | 2017 | 7 | |
| 17 | 2020 | 6 | |
| 18 | 2020 | 4 | |
| 19 | 2017 | 4 | |
| 20 | 2010 | 2 |
About Chisa Okada
Chisa Okada is a scholar working on Molecular Biology, Epidemiology, Surgery, Cancer Research and Neurology, having authored 20 papers that have together received 276 indexed citations. Recurring topics across this work include Autophagy in Disease and Therapy (6 papers), RNA Research and Splicing (3 papers), CRISPR and Genetic Engineering (3 papers), Cancer-related molecular mechanisms research (3 papers), Amyotrophic Lateral Sclerosis Research (2 papers), Gastrointestinal disorders and treatments (2 papers), RNA regulation and disease (2 papers) and Neurogenesis and neuroplasticity mechanisms (1 paper). The work is most often cited by research in Aging (4 citations), Nephrology (13 citations), Physiology (9 citations), Cancer Research (27 citations) and Molecular Biology (121 citations). Chisa Okada has collaborated with scholars based in Japan, Switzerland and Thailand. Frequent co-authors include Hideo Tsukamoto, Noboru Kawabe, Motoki Osawa, Yu Kakimoto, Hitoshi Endo, Masayuki Tatemichi, Satoshi Owada, Akira T. Kawaguchi, Nami Motosugi and Akihiro Umezawa. Their work appears in journals such as PLoS ONE, Anticancer Research, Scientific Reports, Biochemical and Biophysical Research Communications and iScience.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.