Bruce Diaz

1.5k citations
7 papers · 1.2k · h-index 7

Impact in

    • Microtubule and mitosis dynamics
    • Protein Kinase Regulation and GTPase Signaling
    • Melanoma and MAPK Pathways
    • PI3K/AKT/mTOR signaling in cancer
    • 14-3-3 protein interactions
    • Cell death mechanisms and regulation

Papers in

    • Protein Kinase Regulation and GTPase Signaling 6
    • Melanoma and MAPK Pathways 4
    • 14-3-3 protein interactions 2
    • Hippo pathway signaling and YAP/TAZ 2
    • Microtubule and mitosis dynamics 1

Bruce Diaz

7 papers receiving 1.2k citations

Peers

Bruce Diaz
Comparison fields: 5 of 76
  • Cell Biology 281
  • Molecular Biology 948
  • Immunology and Allergy 51
  • Oncology 211
  • Pathology and Forensic Medicine 128
Replace Erik Wilker with:
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Bruce Diaz relative to Erik Wilker United States Erik Wilker's profile →
Citations per field
00.5×1.5×1.8×
Erik Wilker · 1×
Citations per year

Countries citing papers authored by Bruce Diaz

Since Specialization
Citations

This map shows the geographic impact of Bruce Diaz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bruce Diaz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bruce Diaz more than expected).

Fields of papers citing papers by Bruce Diaz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bruce Diaz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bruce Diaz. The network helps show where Bruce Diaz may publish in the future.

Co-authors

The 25 scholars most cited alongside Bruce Diaz, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Bruce Diaz Line = papers co-authored together Bruce Diaz links everyone, so they are left out of the graph.

All Works

7 of 7 papers shown
#Work
1 1998364
2 2003291
3 1997175
4 2001111
5 1997105
6 200586
7 199870

About Bruce Diaz

Bruce Diaz is a scholar working on Molecular Biology, Cell Biology, Pathology and Forensic Medicine, Neurology and Oncology, having authored 7 papers that have together received 1.2k indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (6 papers), Melanoma and MAPK Pathways (4 papers), Hippo pathway signaling and YAP/TAZ (2 papers), 14-3-3 protein interactions (2 papers), Medicinal plant effects and applications (1 paper), Cancer Mechanisms and Therapy (1 paper), NF-κB Signaling Pathways (1 paper) and Microtubule and mitosis dynamics (1 paper). The work is most often cited by research in Cell Biology (281 citations), Molecular Biology (948 citations), Immunology and Allergy (51 citations), Oncology (211 citations) and Pathology and Forensic Medicine (128 citations). Bruce Diaz has collaborated with scholars based in United States. Frequent co-authors include Mark S. Marshall, Darlene Barnard, Alastair J. King, Wenyan Miao, Huaiyu Sun, Shubha Bagrodia, Eva M. Eves, Marsha Rich Rosner, Nicholas Trakul and Kevin C. Corbit. Their work appears in journals such as Molecular and Cellular Biology, The Journal of Experimental Medicine, Journal of Biological Chemistry, Nature and FEBS Letters.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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