Boris Bardot

839 citations
19 papers · 623 · h-index 14

Impact in

Papers in

    • Developmental Biology and Gene Regulation 5
    • RNA modifications and cancer 3
    • Ubiquitin and proteasome pathways 3
    • Cancer-related Molecular Pathways 10

Boris Bardot

18 papers receiving 617 citations

Peers

Boris Bardot
Comparison fields: 5 of 72
  • Immunology and Allergy 54
  • Neurology 95
  • Molecular Biology 430
  • Oncology 161
  • Rheumatology 57
Replace Anita Olofsson with:
Anita Olofsson Sweden
Violaine Closson France
Ludger Klewes Canada
Victoria A. Robb United States
Mateusz Kolanczyk Germany
Aive Åhgren Sweden
Aino Murtomäki United States
Tomoyuki Kawataki Japan
Dave N.T. Aryee Austria
Chiayeng Wang United States
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Citations per field
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Anita Olofsson · 1×
Citations per year

Countries citing papers authored by Boris Bardot

Since Specialization
Citations

This map shows the geographic impact of Boris Bardot's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Boris Bardot with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Boris Bardot more than expected).

Fields of papers citing papers by Boris Bardot

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Boris Bardot. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Boris Bardot. The network helps show where Boris Bardot may publish in the future.

Co-authors

The 25 scholars most cited alongside Boris Bardot, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Boris Bardot Line = papers co-authored together Boris Bardot links everyone, so they are left out of the graph.

All Works

19 of 19 papers shown
#Work
1 2000125
2 200989
3 200473
4 201357
5 201643
6 200243
7 202027
8 200427
9 200425
10 201424
11 200123
12 201220
13 200517
14 201714
15 201311
16 20243
17 20131
18 20241
19 20250

About Boris Bardot

Boris Bardot is a scholar working on Molecular Biology, Oncology, Cell Biology, Physiology and Immunology and Allergy, having authored 19 papers that have together received 623 indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (10 papers), Developmental Biology and Gene Regulation (5 papers), Cell Adhesion Molecules Research (3 papers), Telomeres, Telomerase, and Senescence (3 papers), RNA modifications and cancer (3 papers), Ubiquitin and proteasome pathways (3 papers), Cancer Research and Treatments (2 papers) and Skin and Cellular Biology Research (2 papers). The work is most often cited by research in Immunology and Allergy (54 citations), Neurology (95 citations), Molecular Biology (430 citations), Oncology (161 citations) and Rheumatology (57 citations). Boris Bardot has collaborated with scholars based in France, United States and United Kingdom. Frequent co-authors include Franck Toledo, François Ahimou, Cedric S. Wesley, Emmanuelle Huillard, Maria Marx, Georges Calothy, Sara Jaber, Qingyun Zhang, James Resau and Lilia Topol. Their work appears in journals such as eLife, Oncogene, The Journal of Cell Biology, The International Journal of Developmental Biology and Nature Communications.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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