Beth Rapaport
Impact in
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- BRCA gene mutations in cancer
- Genomic variations and chromosomal abnormalities
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- Genetic factors in colorectal cancer
Papers in
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- DNA Repair Mechanisms 1
- Cell death mechanisms and regulation 1
- Genetics 2
- BRCA gene mutations in cancer 3
- Genomic variations and chromosomal abnormalities 1
- Co-authors
- Kenneth Offit (4 shared papers)Helen J. Huang (3 shared papers)Tomas Kirchhoff (2 shared papers)Prema Kolachana (2 shared papers)Nathan A. Ellis (2 shared papers)Khédoudja Nafa (2 shared papers)Jaya M. Satagopan (2 shared papers)Orit Yossepowitch (2 shared papers)
- Journals
- Journal of the American College of Surgeons (1 paper)Journal of Stroke and Cerebrovascular Diseases (1 paper)Leukemia Research (1 paper)BMC Medical Genetics (1 paper)Leukemia & lymphoma (1 paper)
- Partner nations
- United StatesCanada
In The Last Decade
Beth Rapaport
5 papers receiving 157 citations
Peers
Comparison fields: 5 of 37
- Genetics 89
- Pathology and Forensic Medicine 46
- Cancer Research 27
- Internal Medicine 3
- Molecular Biology 61
Countries citing papers authored by Beth Rapaport
This map shows the geographic impact of Beth Rapaport's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Beth Rapaport with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Beth Rapaport more than expected).
Fields of papers citing papers by Beth Rapaport
This network shows the impact of papers produced by Beth Rapaport. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Beth Rapaport. The network helps show where Beth Rapaport may publish in the future.
Co-authors
The 25 scholars most cited alongside Beth Rapaport, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2003 | 112 | |
| 2 | 2003 | 20 | |
| 3 | 2003 | 13 | |
| 4 | 2010 | 13 | |
| 5 | 2002 | 6 |
About Beth Rapaport
Beth Rapaport is a scholar working on Molecular Biology, Genetics, Pathology and Forensic Medicine, Oncology and Hepatology, having authored 5 papers that have together received 164 indexed citations. Recurring topics across this work include BRCA gene mutations in cancer (3 papers), Cancer-related Molecular Pathways (1 paper), DNA Repair Mechanisms (1 paper), NF-κB Signaling Pathways (1 paper), Genetic factors in colorectal cancer (1 paper), Cell death mechanisms and regulation (1 paper), Genomic variations and chromosomal abnormalities (1 paper) and Hepatocellular Carcinoma Treatment and Prognosis (1 paper). The work is most often cited by research in Genetics (89 citations), Pathology and Forensic Medicine (46 citations), Cancer Research (27 citations), Internal Medicine (3 citations) and Molecular Biology (61 citations). Beth Rapaport has collaborated with scholars based in United States and Canada. Frequent co-authors include Kenneth Offit, Helen J. Huang, Tomas Kirchhoff, Prema Kolachana, Nathan A. Ellis, Khédoudja Nafa, Jaya M. Satagopan, Orit Yossepowitch, Steven B. Johnson and Peter K. Gregersen. Their work appears in journals such as Journal of the American College of Surgeons, Journal of Stroke and Cerebrovascular Diseases, Leukemia Research, BMC Medical Genetics and Leukemia & lymphoma.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.