Takehiro Ono

411 citations
20 papers · 284 · h-index 9

Impact in

Papers in

Takehiro Ono

18 papers receiving 272 citations

Peers

Takehiro Ono
Comparison fields: 5 of 66
  • Endocrinology, Diabetes and Metabolism 76
  • Genetics 67
  • Pathology and Forensic Medicine 39
  • Nephrology 11
  • Molecular Biology 95
Replace Y. Watanabe with:
Y. Watanabe Japan
Taisei Suzuki Japan
Katsuyoshi Kumagai Japan
J. Kühl Germany
M. KOGA Japan
Shinichiro Koyama Japan
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J. Ellis United States
Takehiro Ono relative to Y. Watanabe Japan Y. Watanabe's profile →
Citations per field
00.5×2.8×
Y. Watanabe · 1×
Citations per year

Countries citing papers authored by Takehiro Ono

Since Specialization
Citations

This map shows the geographic impact of Takehiro Ono's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Takehiro Ono with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Takehiro Ono more than expected).

Fields of papers citing papers by Takehiro Ono

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Takehiro Ono. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Takehiro Ono. The network helps show where Takehiro Ono may publish in the future.

Co-authors

The 25 scholars most cited alongside Takehiro Ono, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Takehiro Ono Line = papers co-authored together Takehiro Ono links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 199762
2 199651
3 199847
4 199638
5 201218
6 200816
7 199611
8 201710
9 20099
10 20164
11 19994
12 20113
13 20202
14 20142
15 20112
16 20232
17
Protective effect of FR35447 on experimental cerebral infarction.
19842
18 20141
19 20180
20 20240

About Takehiro Ono

Takehiro Ono is a scholar working on Nuclear and High Energy Physics, Astronomy and Astrophysics, Radiation, Biomedical Engineering and Surgery, having authored 20 papers that have together received 284 indexed citations. Recurring topics across this work include Magnetic confinement fusion research (8 papers), Ionosphere and magnetosphere dynamics (4 papers), Nuclear Physics and Applications (4 papers), Laser-Plasma Interactions and Diagnostics (3 papers), Superconducting Materials and Applications (2 papers), Coagulation, Bradykinin, Polyphosphates, and Angioedema (2 papers), Bone health and treatments (2 papers) and Vitamin D Research Studies (2 papers). The work is most often cited by research in Endocrinology, Diabetes and Metabolism (76 citations), Genetics (67 citations), Pathology and Forensic Medicine (39 citations), Nephrology (11 citations) and Molecular Biology (95 citations). Takehiro Ono has collaborated with scholars based in Japan, South Korea and United States. Frequent co-authors include Yoshiki Seino, Sho Kanzaki, Tomoo Yamate, Subburaman Mohan, David J. Baylink, Toshio Kubo, Hiroyuki Tanaka, Y. Nagai, Hiroyuki Tanaka and Tomoya Nakamura. Their work appears in journals such as Review of Scientific Instruments, Fusion Engineering and Design, Endocrinology, Cytokine and The Journal of Clinical Endocrinology & Metabolism.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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