Kari Alitalo

125.8k citations
724 papers · 99.5k · 44 hit papers · h-index 165

Impact in

Papers in

    • Angiogenesis and VEGF in Cancer 296
    • Lymphatic System and Diseases 277
    • Vascular Tumors and Angiosarcomas 44

Kari Alitalo

716 papers receiving 96.0k citations

Kari Alitalo's Hit Papers

VEGF-C-driven lymphatic drainage enables immunosurveillance of brain tumours 2020 · 380 citations
3800+7+15Years since publication50010001.5k2.0k

Peers

Kari Alitalo
Comparison fields: 5 of 183
  • Oncology 45.1k
  • Immunology and Allergy 4.8k
  • Cancer Research 11.8k
  • Molecular Biology 54.9k
  • Cell Biology 8.5k
Replace Napoleone Ferrara with:
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Citations per field
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Citations per year

Countries citing papers authored by Kari Alitalo

Since Specialization
Citations

This map shows the geographic impact of Kari Alitalo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kari Alitalo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kari Alitalo more than expected).

Fields of papers citing papers by Kari Alitalo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kari Alitalo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kari Alitalo. The network helps show where Kari Alitalo may publish in the future.

Co-authors

The 25 scholars most cited alongside Kari Alitalo, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Kari Alitalo Line = papers co-authored together Kari Alitalo links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 724 papers — load more, or switch the sort, to bring in the rest.

#Work
1
VEGF guides angiogenic sprouting utilizing endothelial tip cell filopodia
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20032308
2
A dural lymphatic vascular system that drains brain interstitial fluid and macromolecules
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20151641
3
Molecular regulation of angiogenesis and lymphangiogenesis
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20071621
4
Induction of tumor lymphangiogenesis by VEGF-C promotes breast cancer metastasis
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20011497
5
A novel vascular endothelial growth factor, VEGF‐C, is a ligand for the Flt4 (VEGFR‐3) and KDR (VEGFR‐2) receptor tyrosine kinases.
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19961439
6
Amplified DNA with limited homology to myc cellular oncogene is shared by human neuroblastoma cell lines and a neuroblastoma tumour
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19831218
7
Expression of the fms-like tyrosine kinase 4 gene becomes restricted to lymphatic endothelium during development.
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19951186
8
Control of vascular morphogenesis and homeostasis through the angiopoietin–Tie system
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20091158
9
Lymphangiogenesis: Molecular Mechanisms and Future Promise
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20101155
10
Vascular endothelial growth factor C is required for sprouting of the first lymphatic vessels from embryonic veins
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20031129
11
Hyperplasia of Lymphatic Vessels in VEGF-C Transgenic Mice
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19971086
12
Lymphangiogenesis in development and human disease
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20051031
13
Vascular endothelial growth factor D (VEGF-D) is a ligand for the tyrosine kinases VEGF receptor 2 (Flk1) and VEGF receptor 3 (Flt4)
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19981007
14
Angiosarcomas Express Mixed Endothelial Phenotypes of Blood and Lymphatic Capillaries
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1999954
15
Clinical applications of angiogenic growth factors and their inhibitors
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1999875
16
The lymphatic vasculature in disease
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2011757
17
Copy number variation and selection during reprogramming to pluripotency
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2011739
18
Cardiovascular Failure in Mouse Embryos Deficient in VEGF Receptor-3
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1998704
19
Homogeneously staining chromosomal regions contain amplified copies of an abundantly expressed cellular oncogene (c-myc) in malignant neuroendocrine cells from a human colon carcinoma.
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1983701
20
The biology of vascular endothelial growth factors
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2005692

About Kari Alitalo

Kari Alitalo is a scholar working on Molecular Biology, Oncology, Cancer Research, Surgery and Cell Biology, having authored 724 papers that have together received 99.5k indexed citations. Recurring topics across this work include Angiogenesis and VEGF in Cancer (296 papers), Lymphatic System and Diseases (277 papers), Cell Adhesion Molecules Research (55 papers), Axon Guidance and Neuronal Signaling (51 papers), Sympathectomy and Hyperhidrosis Treatments (47 papers), Vascular Tumors and Angiosarcomas (44 papers), Vascular Malformations and Hemangiomas (41 papers) and Cancer, Hypoxia, and Metabolism (37 papers). The work is most often cited by research in Oncology (45.1k citations), Immunology and Allergy (4.8k citations), Cancer Research (11.8k citations), Molecular Biology (54.9k citations) and Cell Biology (8.5k citations). Kari Alitalo has collaborated with scholars based in Finland, United States and Sweden. Frequent co-authors include Tuomas Tammela, Michael Jeltsch, Seppo Ylä‐Herttuala, Marika J. Karkkainen, Taija Mäkinen, Terhi Kärpänen, Ralf H. Adams, Arja Kaipainen, Tatiana V. Petrova and Vladimir Joukov. Their work appears in journals such as Blood, Proceedings of the National Academy of Sciences, Circulation Research, Cancer Research and Molecular and Cellular Biology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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