David E. Merriam
Impact in
- Physiology top 2%
- Alzheimer's disease research and treatments
-
- Neuroscience and Neuropharmacology Research
- Nuclear Receptors and Signaling
Papers in
-
- Alzheimer's disease research and treatments 5
-
- Prion Diseases and Protein Misfolding 1
- Cell death mechanisms and regulation 1
- Co-authors
- Wilma Wasco (4 shared papers)Annette C. Crowley (3 shared papers)Joseph D. Buxbaum (3 shared papers)Christina Lilliehöök (2 shared papers)Eun‐Kyoung Choi (2 shared papers)Yuxia Luo (1 shared paper)Olivia G. Hallmark (1 shared paper)Ronald Mancini (1 shared paper)
- Journals
- Nature Medicine (2 papers)Journal of Biological Chemistry (1 paper)Journal of Clinical Investigation (1 paper)Molecular Brain Research (1 paper)
- Partner nations
- United StatesGermanyHong Kong
In The Last Decade
David E. Merriam
5 papers receiving 1.1k citations
Peers
Comparison fields: 5 of 72
- Physiology 766
- Cellular and Molecular Neuroscience 348
- Cell Biology 242
- Pharmacology 187
- Neurology 93
Countries citing papers authored by David E. Merriam
This map shows the geographic impact of David E. Merriam's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David E. Merriam with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David E. Merriam more than expected).
Fields of papers citing papers by David E. Merriam
This network shows the impact of papers produced by David E. Merriam. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David E. Merriam. The network helps show where David E. Merriam may publish in the future.
Co-authors
The 25 scholars most cited alongside David E. Merriam, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 1996 | 481 | |
| 2 | 2000 | 307 | |
| 3 | 1998 | 304 | |
| 4 | 2001 | 56 | |
| 5 | 2001 | 9 |
About David E. Merriam
David E. Merriam is a scholar working on Physiology, Molecular Biology, Pharmacology, Cellular and Molecular Neuroscience and Neurology, having authored 5 papers that have together received 1.2k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (5 papers), Cholinesterase and Neurodegenerative Diseases (2 papers), Computational Drug Discovery Methods (1 paper), Trace Elements in Health (1 paper), Nuclear Receptors and Signaling (1 paper), Parkinson's Disease Mechanisms and Treatments (1 paper), Prion Diseases and Protein Misfolding (1 paper) and Cell death mechanisms and regulation (1 paper). The work is most often cited by research in Physiology (766 citations), Cellular and Molecular Neuroscience (348 citations), Cell Biology (242 citations), Pharmacology (187 citations) and Neurology (93 citations). David E. Merriam has collaborated with scholars based in United States, Germany and Hong Kong. Frequent co-authors include Wilma Wasco, Annette C. Crowley, Joseph D. Buxbaum, Christina Lilliehöök, Eun‐Kyoung Choi, Yuxia Luo, Olivia G. Hallmark, Ronald Mancini, R.E. Tanzi and Robert D. Moir. Their work appears in journals such as Nature Medicine, Journal of Biological Chemistry, Journal of Clinical Investigation and Molecular Brain Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.