Woodring E. Wright
Impact in
- Aging top 0.01%
- Genetics, Aging, and Longevity in Model Organisms
- Physiology top 0.01%
- Telomeres, Telomerase, and Senescence
Papers in
-
- Advanced biosensing and bioanalysis techniques 45
- Muscle Physiology and Disorders 44
- RNA Interference and Gene Delivery 43
- DNA Repair Mechanisms 32
- CRISPR and Genetic Engineering 20
- Physiology 187
- Telomeres, Telomerase, and Senescence 184
- Co-authors
- Jerry W. Shay (210 shared papers)Mieczyslaw A. Piatyszek (11 shared papers)Calvin B. Harley (8 shared papers)Shawn E. Holt (12 shared papers)Scott L. Weinrich (5 shared papers)Nam W. Kim (4 shared papers)Michael D. West (4 shared papers)Karen R. Prowse (3 shared papers)
- Journals
- Molecular and Cellular Biology (20 papers)Experimental Cell Research (10 papers)Journal of Biological Chemistry (10 papers)Oncogene (9 papers)Proceedings of the National Academy of Sciences (9 papers)
- Partner nations
- United StatesSaudi ArabiaFrance
In The Last Decade
Woodring E. Wright
294 papers receiving 42.7k citations
Woodring E. Wright's Hit Papers
Peers
Comparison fields: 5 of 172
- Aging 4.2k
- Physiology 24.1k
- Biotechnology 3.6k
- Molecular Biology 26.8k
- Genetics 2.4k
Countries citing papers authored by Woodring E. Wright
This map shows the geographic impact of Woodring E. Wright's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Woodring E. Wright with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Woodring E. Wright more than expected).
Fields of papers citing papers by Woodring E. Wright
This network shows the impact of papers produced by Woodring E. Wright. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Woodring E. Wright. The network helps show where Woodring E. Wright may publish in the future.
Co-authors
The 25 scholars most cited alongside Woodring E. Wright, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 294 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Specific Association of Human Telomerase Activity with Immortal Cells and Cancer Hit paper breakdown → | 1994 | 5994 |
| 2 | Extension of Life-Span by Introduction of Telomerase into Normal Human Cells Hit paper breakdown → | 1998 | 3911 |
| 3 | Myogenin, a factor regulating myogenesis, has a domain homologous to MyoD Hit paper breakdown → | 1989 | 1149 |
| 4 | Telomerase activity in human germline and embryonic tissues and cells Hit paper breakdown → | 1996 | 1099 |
| 5 | Reconstitution of human telomerase with the template RNA component hTR and the catalytic protein subunit hTRT Hit paper breakdown → | 1997 | 812 |
| 6 | Functional activity of myogenic HLH proteins requires hetero-oligomerization with E12/E47-like proteins in vivo Hit paper breakdown → | 1991 | 780 |
| 7 | Pericytes of human skeletal muscle are myogenic precursors distinct from satellite cells Hit paper breakdown → | 2007 | 741 |
| 8 | Human Telomerase and Its Regulation Hit paper breakdown → | 2002 | 696 |
| 9 | A transcriptionally active DNA-binding site for human p53 protein complexes. Hit paper breakdown → | 1992 | 644 |
| 10 | Absence of cancer–associated changes in human fibroblasts immortalized with telomerase Hit paper breakdown → | 1999 | 639 |
| 11 | Telomeres and telomerase: three decades of progress Hit paper breakdown → | 2019 | 625 |
| 12 | Normal human chromosomes have long G-rich telomeric overhangs at one end Hit paper breakdown → | 1997 | 590 |
| 13 | Expression of two myogenic regulatory factors myogenin and MyoDl during mouse embryogenesis Hit paper breakdown → | 1989 | 584 |
| 14 | Senescence and immortalization: role of telomeres and telomerase Hit paper breakdown → | 2004 | 567 |
| 15 | Hayflick, his limit, and cellular ageing Hit paper breakdown → | 2000 | 521 |
| 16 | 1999 | 452 | |
| 17 | 1999 | 441 | |
| 18 | 1995 | 434 | |
| 19 | 1989 | 429 | |
| 20 | 1989 | 385 |
About Woodring E. Wright
Woodring E. Wright is a scholar working on Molecular Biology, Physiology, Aging, Oncology and Biotechnology, having authored 294 papers that have together received 43.7k indexed citations. Recurring topics across this work include Telomeres, Telomerase, and Senescence (184 papers), Advanced biosensing and bioanalysis techniques (45 papers), Muscle Physiology and Disorders (44 papers), RNA Interference and Gene Delivery (43 papers), Genetics, Aging, and Longevity in Model Organisms (39 papers), DNA Repair Mechanisms (32 papers), Cancer Research and Treatments (24 papers) and CRISPR and Genetic Engineering (20 papers). The work is most often cited by research in Aging (4.2k citations), Physiology (24.1k citations), Biotechnology (3.6k citations), Molecular Biology (26.8k citations) and Genetics (2.4k citations). Woodring E. Wright has collaborated with scholars based in United States, Saudi Arabia and France. Frequent co-authors include Jerry W. Shay, Mieczyslaw A. Piatyszek, Calvin B. Harley, Shawn E. Holt, Scott L. Weinrich, Nam W. Kim, Michael D. West, Karen R. Prowse, Gregg B. Morin and Victor K. Lin. Their work appears in journals such as Molecular and Cellular Biology, Experimental Cell Research, Journal of Biological Chemistry, Oncogene and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.