A. Briddon

25 papers receiving 454 citations

Peers

A. Briddon
Comparison fields: 5 of 70
  • Clinical Biochemistry 199
  • Biochemistry 96
  • Rheumatology 104
  • Gastroenterology 37
  • Immunology and Allergy 17
Replace M. A. H. Giesberts with:
M. A. H. Giesberts Netherlands
J. M. van der Klei‐van Moorsel Netherlands
Vladimir Sarnavka Croatia
L Cathelineau France
Muriel Quillard France
S. K. Hall United Kingdom
Floris C. Hofstede Netherlands
Louise M. Pepe United States
François‐Guillaume Debray Belgium
Jun Ye China
A. Briddon relative to M. A. H. Giesberts Netherlands M. A. H. Giesberts's profile →
Citations per field
00.5×
M. A. H. Giesberts · 1×
Citations per year

Countries citing papers authored by A. Briddon

Since Specialization
Citations

This map shows the geographic impact of A. Briddon's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by A. Briddon with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites A. Briddon more than expected).

Fields of papers citing papers by A. Briddon

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by A. Briddon. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by A. Briddon. The network helps show where A. Briddon may publish in the future.

Co-authors

The 25 scholars most cited alongside A. Briddon, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with A. Briddon Line = papers co-authored together A. Briddon links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 25 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2005211
2 198465
3 199817
4 200217
5 200316
6 200615
7 197815
8 200314
9 198412
10 199611
11 20108
12 20108
13 19867
14 19807
15 20016
16 19876
17 19866
18 19965
19 20035
20 19914

About A. Briddon

A. Briddon is a scholar working on Clinical Biochemistry, Molecular Biology, Rheumatology, Biochemistry and Surgery, having authored 25 papers that have together received 465 indexed citations. Recurring topics across this work include Metabolism and Genetic Disorders (11 papers), Folate and B Vitamins Research (7 papers), Amino Acid Enzymes and Metabolism (5 papers), Esophageal and GI Pathology (3 papers), Gout, Hyperuricemia, Uric Acid (2 papers), Celiac Disease Research and Management (2 papers), Iron Metabolism and Disorders (2 papers) and Renal function and acid-base balance (2 papers). The work is most often cited by research in Clinical Biochemistry (199 citations), Biochemistry (96 citations), Rheumatology (104 citations), Gastroenterology (37 citations) and Immunology and Allergy (17 citations). A. Briddon has collaborated with scholars based in United Kingdom, Netherlands and Italy. Frequent co-authors include Simon Heales, Alan D. Phillips, Carlos Maluenda, J A Walker‐Smith, Johannes Zschocke, Robert Surtees, Clare Beesley, Peter T. Clayton, Philippa B. Mills and M. P. Champion. Their work appears in journals such as Amino Acids, Journal of Inherited Metabolic Disease, Annals of Clinical Biochemistry International Journal of Laboratory Medicine, Journal of Pediatric Gastroenterology and Nutrition and Clinica Chimica Acta.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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