Autophagy Is Activated for Cell Survival after Endoplasmic ReticulumStress
Impact in
- Epidemiology 1.1k
- Cell Biology 829
Classified as
- Journal
- Molecular and Cellular Biology
In The Last Decade
doi.org/10.1128/mcb.01453-06 →Countries where authors are citing Autophagy Is Activated for Cell Survival after Endoplasmic ReticulumStress
This map shows the geographic impact of Autophagy Is Activated for Cell Survival after Endoplasmic ReticulumStress. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Autophagy Is Activated for Cell Survival after Endoplasmic ReticulumStress with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Autophagy Is Activated for Cell Survival after Endoplasmic ReticulumStress more than expected).
Fields of papers citing Autophagy Is Activated for Cell Survival after Endoplasmic ReticulumStress
This network shows the impact of Autophagy Is Activated for Cell Survival after Endoplasmic ReticulumStress. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the Autophagy Is Activated for Cell Survival after Endoplasmic ReticulumStress.
About Autophagy Is Activated for Cell Survival after Endoplasmic ReticulumStress
This paper, published in 2006, received 1.6k indexed citations . Written by Maiko Ogata, Shin‐ichiro Hino, Atsushi Saito, Keisuke Morikawa, Shinichi Kondo, Soshi Kanemoto, Tomohiko Murakami, Manabu Taniguchi, Ichiro Tanii and Kazuya Yoshinaga covering the research area of Epidemiology and Cell Biology. It is primarily cited by scholars working on Epidemiology (1.1k citations), Cell Biology (829 citations), Molecular Biology (526 citations), Surgery (156 citations) and Physiology (145 citations). Published in Molecular and Cellular Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
This paper is also available at doi.org/10.1128/mcb.01453-06.